Free Course Image Renal Pathology

Free online courseRenal Pathology

Duration of the online course: 14 hours and 6 minutes

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Build clinical confidence with this free renal pathology course—master ABG interpretation, electrolyte disorders, AKI/CKD and kidney stones, plus quizzes and certificate.

In this free course, learn about

  • Principles of acid–base disorders and stepwise ABG interpretation
  • Normal blood pH range and meaning of acidemia when pH is low
  • Respiratory acidosis: causes, ABG patterns, and physiologic compensation
  • Control of ventilation: CO2/H+ sensing driving medullary respiratory output
  • Respiratory alkalosis: causes (e.g., salicylates) and ABG patterns
  • Metabolic alkalosis: key causes incl. volume depletion and HCO3− retention
  • Metabolic acidosis: anion gap vs non–anion gap and common etiologies
  • Acute kidney injury: prerenal/intrinsic/postrenal causes, diagnosis, treatment
  • Chronic kidney disease: reduced GFR, kidney roles affected, management basics
  • Hyperkalemia: drivers (↓excretion, shifts, excess load), ECG risks, treatment
  • Hyponatremia: volume/ADH physiology, evaluation approach, safe correction
  • Hypernatremia: water deficit physiology and compensation via thirst/ADH
  • Nephrolithiasis: stone types, risk factors, and citrate’s protective role

Course Description

Renal pathology sits at the center of everyday clinical decision-making because the kidneys quietly control acid-base balance, electrolyte homeostasis, blood pressure regulation, and fluid status. When these systems drift off course, the result can be a subtle lab abnormality, a rapid deterioration in a sick patient, or a chronic decline that changes someone’s life. This free online course is designed to help you connect physiology to real-world interpretation so you can approach kidney-related problems with clarity and speed.

You will build a practical framework for reading arterial blood gases and recognizing acid-base disorders, learning how respiratory and metabolic disturbances arise and how compensation works. From there, the course strengthens your ability to reason through common electrolyte problems, including potassium and sodium derangements, focusing on why they happen, what clinical features to watch for, and how treatment choices map to underlying mechanisms. The goal is not memorization, but pattern recognition anchored in physiology so that results on the screen translate into informed next steps at the bedside.

The course also tackles acute and chronic kidney disease with attention to etiology, pathophysiology, diagnostic thinking, and initial management considerations. You will learn how reduced effective arterial blood volume can impair perfusion, why glomerular filtration rate matters, and how kidney dysfunction amplifies acid-base and electrolyte instability. Finally, you will explore nephrolithiasis, including risk factors and the biologic changes that promote stone formation, helping you understand prevention and recurrence from a mechanistic perspective.

Throughout, short exercises reinforce key concepts and keep you actively engaged, making it easier to retain what you learn and apply it during study, clinical rotations, or practice. If you want a cohesive, physiology-first way to sharpen interpretation skills and improve renal-related clinical reasoning, this course provides a structured and approachable path forward.

Course content

  • Video class: Acid Base Disorders and ABG Interpretation | Introduction 58m
  • Exercise: What is the normal pH range of the human blood, and what condition is indicated when the blood pH is below this range?
  • Video class: Respiratory Acidosis 41m
  • Exercise: What physiological mechanism is primarily responsible for regulating the firing of neurons that control breathing, such as the diaphragm and intercostal muscles?
  • Video class: Respiratory Alkalosis 32m
  • Exercise: Which of the following is a common cause of respiratory alkalosis characterized by a common drug hyperstimulating the medullary respiratory centers?
  • Video class: Metabolic Alkalosis 40m
  • Exercise: What is a common cause of metabolic alkalosis related to volume depletion and an increase in bicarbonate reabsorption?
  • Video class: Metabolic Acidosis 1h05m
  • Exercise: In the video, two types of metabolic acidosis are mentioned. Which of the following is NOT a cause of elevated anion gap metabolic acidosis (AGMA)?
  • Video class: Acute Kidney Injury (AKI) | Etiology, Pathophysiology, Clinical Features, Diagnosis, Treatment 1h49m
  • Exercise: Which of the following is a potential cause of pre-renal acute kidney injury due to reduced effective arterial blood volume with a normal total body volume?
  • Video class: Chronic Kidney Disease (CKD) | Etiology, Pathophysiology, Clinical Features, Diagnosis, Treatment 1h25m
  • Exercise: What is the primary role of the kidneys that can be affected in chronic kidney disease (CKD), leading to reduced glomerular filtration rate (GFR)?
  • Video class: Hyperkalemia | Etiology, Pathophysiology, Clinical Features, Diagnosis, Treatment 1h02m
  • Exercise: What are the major contributors to the increase in blood potassium levels in hyperkalemia?
  • Video class: Hyponatremia 3h09m
  • Exercise: Which of the following mechanisms primarily triggers the release of antidiuretic hormone (ADH) in response to decreased effective arterial blood volume?
  • Video class: Hypernatremia 1h14m
  • Exercise: What is the primary compensation mechanism for hypernatremia when serum sodium levels are elevated?
  • Video class: Nephrolithiasis | Kidney Stones 1h27m
  • Exercise: In the pathophysiology of nephrolithiasis, which condition is associated with a decrease in urinary citrate leading to an increased risk of calcium oxalate stone formation?

This free course includes:

14 hours and 6 minutes of online video course

Digital certificate of course completion (Free)

Exercises to train your knowledge

100% free, from content to certificate

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